gum diseaseWord格式文档下载.docx
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gum diseaseWord格式文档下载.docx
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Acutegumpapillitis
Druggumhyperplasia
Thediseaseofgumfibrosis
Thegumdiseaseofleukemiaisdamaged
Acutemultiplegumsabscess
Lowgumtumour
Anecroticulcerativegumdisease
VitaminCdeficiencyofgingivitis
GingivitisGingivitisisonlyassociatedwithdentalplaque
BorderlineGingivitis
SimpleGingivitisSimpleGingivitis
A.nootherlocalstimulant
B.accompaniedbylocalstimulant
Prevalenceiswidespread
Theprevalenceofchildrenandadolescentsis70-90%
Pubertypeaks
Thecauseof
Plaque-initiatingfactor
Dentalrock,foodembedded,badrepairbody-localpromotionfactors
Clinicalpathology
Initiallesionwasinitiallesion
Time:
2-4days
Clinical:
canbehealthygums
Pathology:
vasodilation,gingivalsulcusfluid(GCF),whitebloodcells(mainlyforthePMNs)bleedingfromthewalltothecombinationofepitheliumandgingivalsulcuszone,inflammatoryinfiltrationareaaccountsforabout5%oftheconnectivetissue
Earlylesionsearlylesion
4-7days
thegumsareredandswollen
angiogenesisexpands.Mainlylymphocyte(about75%),andneutrophilsinfiltrate,inflammationis15%,collagenisdestroyed,whitebloodcellsdevourbacteria,epitheliumisstapled.
Establishanestablishedlesion
chronicguminflammation
thegumsareclearlyinflamed,redandswollen,andthebleedingofthegingivadeepens
plasmacells(Page&
Schroeder),venousreturn,gumhypoxia,inflammationarealossofcollagen,leukocyteinfiltration,epithelialspikessignificantly,buttheepithelialattachmentpositionunchanged
orthodontia
Clinicalmanifestationsof
Thegum
pink
Theshapeisthinandthesurfaceofthetoothisalittlemorecolorful
It'
sdenseandresilient
Thechannelisnotmorethantwotothreemmdeep
Theattachmentofthepositionoftheboneinthebone
nosediagnosis
Darkred,redandred
Thegingivaisthickenedanddoesnotclingtothenipple
Thesoftandfragilenatureisinelastic
Thedepthofgingiviescanbeincreasedbymorethan3mmtoformapseudoperiodontalpouch
Theimportantobjectiveindicatorsofthediagnosisanddiagnosisofguminflammationwerediagnosed
uvioglya
Self-conscioussymptomsofbrushingandbitingarebleeding
Diagnosticlocalfactorsandclinicalmanifestations
Differentialdiagnosis:
Earlyperiodontitis:
attachmentloss,boneabsorptionofbone
Blooddisease
Necroticulcerativegumdisease
HIVrelatedgingivitis
VitaminCDeficiencyofGingivitisGingivitisinVitaminCDeficiency
Cause:
thelackofVitaminCthatgingivalconnectivetissueedema,hemorrhageandcollagendenaturation,causegumdefenseresponsesagainstnormalbacteriaisrestrained,accentuateinflammation,gingivalhyperplasia.
Clinicalmanifestation:
gingivium,darkred,soft,fragile,surfacelight,spontaneousorlightcontacthemorrhage,commonhavesurfacenecrosis,pseudofilmformation.
gumchronicinflammation,inflammationcellinfiltrates.Capillarydilation,extensivebleeding.Edema,diffuse,collagendegeneration,Collagenfibersandfibroblastsarerare.
Therapeuticprinciples
Removetheetiologicaloralhygieneguidelines(OHI)
Cleans(clearingplaqueandgum)
Correctplaqueretentionfactors(foodinlaid,badrepair,etc.)
Prognosisandrepossession
Timelytreatment-goodprognosis
Aftertreatmenttheplaquecontrolisnotgood-gingivitisrelapses
Untreated--gingitis--periodontitis
Prevention:
OralhygieneinstructionOHI,therightwaytobrushyourteeth,thoroughlycontroltheplaque
Regularperiodontalexamination
HyperplasticGingivitis
Andchronicgingivitis
Mouthbreathing
occlusion
Goodhairontheupperandlowerfrontteeth
Inflammatoryswelling(granulation)
Thegumsareredandsoftandbrightandeasytobleed
Papillaryglobulia
Substantialhypertrophy(fibretype)
Thegumbleedsreducethetextureandthetextureismoreelastic
Thepathological
Inflammatorygranulationtissue
Capillarydilationandcongestion
Alargenumberofinflammatorycellsinfiltrateandooze
Epithelium,connectivetissuehyperplasia
Largeamountsoffibroblastsandnewborncollagenbundles
Thediagnosis
Age,location,andtypicalperformance
Removealllocalstimulitocontrolplaque
Adjuvanttherapy
Surgicalgumoplasty
PregnancyGingivitis(PregnancyG
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